Mechanics has emerged as a necessary discipline to understand embryogenesis. The effect of mechanical forces in tissue development and cell fate determination has been demonstrated, showing that cells respond to forces and mechanical cues.The Drosophila melanogaster is a model organism to study mechanics, with studies showing the mechanical control of cell shape and morphogenesis. Recently, the actomyosin cortex has attracted attention as a key regulator of cell mechanics. In this work, the morphogenetic process of dorsal closure is chosen to study the mechanical effect of actomyosin pulsations during development.Dorsal closure involves the closing of an epidermal gap in the embryo by the contraction of the amnioserosa, the extra-embryonic tissue covering the gap. During this time, cells delaminate from the amnioserosa, but there are no intercalations, migrations or divisions, making it an ideal system to study pulsatile actomyosin, or foci. Biophysical modelling suggests various modes of focus' motion such as advection or diffusion, but the kinetics of these foci are poorly understood. Thus, this work offers the mechanistical quantification of focus behaviour with a stress on kinetic properties.To that end, we use image analysis to create a tracking algorithm for travelling Myosin pulses. In four chapters, the thesis describes data processing tools which characterize focus behaviour, analysing a set of amnioserosa time-lapse movies.In chapter 1, the dataset of two channel (E-cadherin and Myosin II) time-lapse movies of the amnioserosa are described. Using the first channel, the cell membranes are identified. On the second channel, a method of pre-processing is defined, necessary for uniform focus identification over the whole dataset. Through this, the focus static properties, such as characteristic sizes and distribution on cell surface, are quantified.In chapter 2, the introduction of tracking algorithm enables temporal linkages among the identified foci. Merging and splitting behaviour of foci is seen. Under a point-particle ansatz, focus' kinematic properties, such as speeds, durations and angles of deviations between subsequent steps, are described. It is noted that the angles of deviation are non-isotropic, indicating directional motion. It is observed that foci always have non-zero speed between frames, suggesting motion that is not purely diffusive.In chapter 3, through further point-particle analysis of the trajectories of foci, their mean-squared-distances are quantified. Fitting a power-law, the median exponent is found to be in the super-diffusive regime of motion. Though impeded by the small duration of trajectories, this is consistent with the hypothesis of a self-avoiding motion. Measuring the mean direction of trajectory, it is seen that the orientation of the individual steps is preferentially aligned according to this direction. This is found to be linked to cellular confinement due to anisotropy in cell shapes.In chapter 4, the continuous Myosin signal is analysed, and the apical features of florescence in the known spatio-temporal neighbourhoods of foci are visualized through kymographs. These are then averaged to look at the properties of the apical Myosin signal in the regions where a focus has been, and will be. We find the average kinetics of a focus is followed by the phenomenon of Myosin depletion around it. We also note the presence of high Myosin signal across cell-membranes from foci.The work posits a model of self-avoidance due to substrate refractoriness as a mechanism for focus propagation and death. High myosin concentration at a sub-cellular region would be followed by a local refractor